Transient ST Segment Elevation Caused by Intracoronary Thrombus after Acute Carbon Monoxide Poisoning

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dc.authoridTURGAY YILDIRIM, OZGE/0000-0002-6731-4958
dc.authoridAksit, Ercan/0000-0002-4478-4324
dc.authoridBARDAKCI, OKAN/0000-0001-6829-7435
dc.contributor.authorAksit, Ercan
dc.contributor.authorYildirim, Ozge Turgay
dc.contributor.authorAydin, Fatih
dc.contributor.authorBardakci, Okan
dc.contributor.authorAydin, Ayse Huseyinoglu
dc.date.accessioned2025-01-27T20:46:03Z
dc.date.available2025-01-27T20:46:03Z
dc.date.issued2019
dc.departmentÇanakkale Onsekiz Mart Üniversitesi
dc.description.abstractCarbon monoxide (CO) poisoning is the most common cause of death and injury among all poisonings. Myocardial injury is detected in one-third of CO poisonings. In this Case Report, a previously healthy 41-year-old man was referred for CO poisoning. The initial electrocardiogram (ECG) showed 1 mm ST segment elevation in leads DII, DIII, and aVF. As the patient did not describe chest pain and had no cardiac symptoms, ECG was repeated 10 minutes later and it was seen that ST segment elevation disappeared. As the patient had a transient ST segment elevation and elevated high-sensitive Tn-T (HsTn-T), the patient was transferred to the coronary angiography laboratory. The patient's left coronary system was normal, but a thrombus image narrowing the lumen by approximately 60% was observed in the right coronary artery. Intravenous tirofiban was administered for 48 hours. Control coronary angiography showed continuing thrombus formation and a bare metal stent was successfully implanted. This is the first reported case with transient ST segment elevation associated with acute coronary thrombus caused by CO poisoning. It may be recommended that patients with CO poisoning should be followed-up with a 12-lead ECG monitor or 24-hour ECG Holter monitoring, even if they show no cardiac symptoms and echocardiography shows no wall motion abnormality. Early coronary angiography upon detection of such dynamic ECG changes in these recordings as ST segment elevation can reduce the risk of myocardial infarction (MI) and mortality in these patients.
dc.identifier.doi10.1017/S1049023X19004898
dc.identifier.endpage680
dc.identifier.issn1049-023X
dc.identifier.issn1945-1938
dc.identifier.issue6
dc.identifier.pmid31597072
dc.identifier.scopus2-s2.0-85073354458
dc.identifier.scopusqualityQ1
dc.identifier.startpage677
dc.identifier.urihttps://doi.org/10.1017/S1049023X19004898
dc.identifier.urihttps://hdl.handle.net/20.500.12428/24796
dc.identifier.volume34
dc.identifier.wosWOS:000513131000016
dc.identifier.wosqualityQ3
dc.indekslendigikaynakWeb of Science
dc.indekslendigikaynakScopus
dc.indekslendigikaynakPubMed
dc.language.isoen
dc.publisherCambridge Univ Press
dc.relation.ispartofPrehospital and Disaster Medicine
dc.relation.publicationcategoryinfo:eu-repo/semantics/openAccess
dc.rightsinfo:eu-repo/semantics/closedAccess
dc.snmzKA_WoS_20250125
dc.subjectangioplasty
dc.subjectcarbon monoxide
dc.subjectcoronary thrombosis
dc.subjectmyocardial infraction
dc.subjectpoisoning
dc.titleTransient ST Segment Elevation Caused by Intracoronary Thrombus after Acute Carbon Monoxide Poisoning
dc.typeArticle

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