Predictors of coronary collaterals in patients with non ST-ellevated acute coronary syndrome: the paradox of the leukocytes

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dc.authoridTENEKECIOGLU, ERHAN/0000-0003-4376-2833
dc.contributor.authorTenekecioglu, Erhan
dc.contributor.authorYilmaz, Mustafa
dc.contributor.authorKaraagac, Kemal
dc.contributor.authorBekler, Adem
dc.contributor.authorAslan, Burhan
dc.contributor.authorDemir, Serafettin
dc.contributor.authorKuzeytemiz, Mustafa
dc.date.accessioned2025-01-27T20:29:01Z
dc.date.available2025-01-27T20:29:01Z
dc.date.issued2014
dc.departmentÇanakkale Onsekiz Mart Üniversitesi
dc.description.abstractAim of the study: Atherosclerosis represents active inflammation in which leukocytes play significant role. Coronary collateral development is a response to myocardial ischaemia. In this study we aimed to investigate the association of the leukocytes with coronary collateral development in patients with non ST-elevated acute coronary syndromes (NST-ACS). Material and methods: A total of 251 consecutive patients were hospitalized in our hospital with a diagnosis of NST-ACS. The blood samples were collected 1-hour after admission to the hospital and peripheral leukocytes (neutrophils, monocytes and lymphocytes) were examined. All patients underwent coronary angiography. The coronary collateral vessels (CCV) are graded according to the Rentrop scoring system. Results: Group 1 consisted of 146 patients with Rentrop 0 and Group 2 consisted of 105 patients with Rentrop I, 2 and 3. The presence of CCV was significantly associated with neutrophil count, lymphocyte count, monocyte count and neutrophil-lymphocyte ratio (NLR). In subgroup analyses, higher NLR was significantly associated with good CCV development in patients with NST-ACS. Conclusions: Higher neutrophil count, monocyte count and NLR and lower lymphocyte count on admission, were associated with the presence of CCV in patients with NST-ACS. High NLR may predict good collateral development in patients with NST-ACS.
dc.identifier.doi10.5114/ceji.2014.42130
dc.identifier.endpage90
dc.identifier.issn1426-3912
dc.identifier.issn1644-4124
dc.identifier.issue1
dc.identifier.pmid26155105
dc.identifier.scopus2-s2.0-84900544159
dc.identifier.scopusqualityQ3
dc.identifier.startpage83
dc.identifier.urihttps://doi.org/10.5114/ceji.2014.42130
dc.identifier.urihttps://hdl.handle.net/20.500.12428/22779
dc.identifier.volume39
dc.identifier.wosWOS:000336515500015
dc.identifier.wosqualityQ4
dc.indekslendigikaynakWeb of Science
dc.indekslendigikaynakScopus
dc.indekslendigikaynakPubMed
dc.language.isoen
dc.publisherTermedia Publishing House Ltd
dc.relation.ispartofCentral European Journal of Immunology
dc.relation.publicationcategoryinfo:eu-repo/semantics/openAccess
dc.rightsinfo:eu-repo/semantics/openAccess
dc.snmzKA_WoS_20250125
dc.subjectleukocytes
dc.subjectangiogenesis
dc.subjectcoronary collateral vessel
dc.titlePredictors of coronary collaterals in patients with non ST-ellevated acute coronary syndrome: the paradox of the leukocytes
dc.typeArticle

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