Potential effects of sCD40L, CD36, IL-23 and arginase-1 molecules on the pathogenesis of brucellosis

Kızmaz, Muhammed AliEllergezen, Pınar HızDemir, NesrinCagan, ErenÇolak, ZehranurAkalın, Emin HalisOral, Haluk BarbarosBudak, Ferah2023-03-142023-03-1420210014-29801521-4141https://hdl.handle.net/20.500.12428/3806Brucellosis is a systemic infectious disease that can be transmitted from animals to humans, ranging from mild to severe clinical pictures. In our study, we aimed to reveal the roles of sCD40L, CD36, IL‐23 and arginase‐1 (ARG1) molecules in the pathogenesis of brucellosis. sCD40L binds and activates CD40 on antigen‐presenting cells, thereby promoting the secretion of pro‐inflammatory cytokines and nitric oxide (NO) synthesis.eninfo:eu-repo/semantics/openAccessBacterial infectionsEffector moleculesImmune response tracingInfectious diseasePotential effects of sCD40L, CD36, IL-23 and arginase-1 molecules on the pathogenesis of brucellosisConference Object51Supp1319319Q2WOS:000753366401509